I’ve chosen to divide this article into two segments. The first, an introductory one, will discuss Cuntster’s recent impudent remarks on the geopolitical situation in the DPRK. That, as trivial a remark as it may seem, he confused ‘microcosm’ with ‘macrocosm’ is very telling of the sum-totality of ignorance which should compel anyone to shove the fecal fudge advice of “acting the authority without place” back up his own rectum, of course, it probably wouldn’t perturb the petulant, pestilential faggot-in-a-dress in the slightest.
Far more representative of authoritarian fantasies in the metaphorical is the current state in which his regime mascarades a democracy in extension of fully enfranchised rights to everyone. The medicalization and desocialization of autists perpetrated upon them from the earliest of days here, stands in stark contrast to the non-recognition of autism by Choson, in which many of the better ones will be accorded full rights, and the worst, sent to 49s. If it was a gamble I was, and I am, prepared to take, I’d play roulette with the “north” Koreans any day, knowing that, absent of deliberately conspired desocialization, I’d be spared of the 49 fate in the same way I wouldn’t be the predations of the NHS.
Brave New World Order, as encapsulated by David Chac and further elucidated in The Wisdom of Autphag, is a perfect descriptor for the circumstances in which the autist are now enshrouded, and not by nature; demelioration of IQc’s contrivance, in contrariety to the most genetically fit reality, only foments a strawman allowing petulant little childish faggots with immature etho-moral conceptions like Cuntster to run with them a while, all at the cost of the autist, ever aggressed against in this fashion, losing their colour.
Meanwhile, the state is ever hard at work to conceal Cuntster’s negritude with opportunities that by the rules of nature should rightfully be unafforded to him.
It really irks me to see Cuntster cite situations which have long since seen resolution, which he didn’t look at the GNP data for, the GNP/capita data, the communist equivalent measure of whole manufacturing production units, that per capita, calorific consumption per capita, height growth, or even his favourite measure, “let’s see YouTube videos and pretend it’s science.” He didn’t even restate, and so therefore reliably-in-likelihood never visited, either the history from the revolutionary perspective, or the Western perspective (a fusion of perspectives is actually available from a fairly neutrally written CIA country study, though it is already a decade out of date, with the next edition due for some time mid-next decade). He speaks about the multiplicity of aid supposedly being received by the country (amounting to less than 0.25% of the GDP officially, at only low-hundreds of millions USD; the DPRK earns that alone with a single shipment of arms), yet ignores the embargo (which only China has really ignored and many South East Asian countries are only ignorant of due to ineffectual bureaucracies), and the achievements of Chosun to spite it (50% of its GDP recovered in a decade back to pre-collapse levels, and, post-2007, an effective doubling of the size of the economy over in certain areas; agriculture is now at the maximum extent of self-sufficiency, despite arable land limitations, thanks to aquaculture and processed food innovations, and food-intake has returned to near pre-collapse levels). Instead, his thought process went something on the lines of, “let’s infuriate ‘Autphag’ with BBC-lifted platitudes, sprinkled with my usual angrily adjectivally-laden sass, feigned indignation, and pseudomoral parsimony, and let’s see where we go with this.”
Even with the lattermost measure, I can see, the youngest 2017 army recruits were, hmm, maybe about my height (5’8”) actually. This is a vast improvement from the 1998 videos, in which they were visibly smaller than somebody like, say, even my step-father, a measly 5’5” (i.e. the average “north” Korean recruit during the Arduous March was smaller than that, and has grown, on average, maybe half a foot). That doesn’t look like a cohort being starved to death, and these divisions are drawn in recruitment nationwide, debunking the “Pyongyang is privileged” argument.
There is nothing like, nor was there ever even at the worst severest periods in Korean history, anything like “99% of the population starving to death.”
Yet, he is one to speak of the arrogance of superimposing a view of how the world should work, yet expects to successfully superimpose the direct translation in empirical findings of a populationally incompatible cohort upon an introspection he denies attribution of me; the same arrogance from which stems that the Korean populace should ever be expected to swallow his hubris-fuelled way of life at their expense. That’s where one should derive their real disgust. Then, speaking so condescendingly of the Kim dynasty without qualification as to the observation of their true talent, probably surpassing this pseudo-negress’s by miles.
His disgusting pretences to sanctimony are then encapsulated to their epochal peak of consummation when he contents himself with his utter dissympathy for the autist’s predicament at home, enough to feel an unspoken, self-satisfied glee at their suffering, whilst expressing feigned indignation at that he supposes is similarly happening 12,000 miles away from him. We know where his true anger, then, stems; that he hasn’t enough autists to push to the brink of ruin.
Social reptoid perfectly describes his social attitudes. They are unoriginal to him and a pathetically transparent reflection of the environs surrounding him, with no backing conviction rooted in anything likable to “compassion”; how could a position of lamenting the woes of starved, outer-periphery Koreans be correctly reconciled as an organic statement when his internalised sentiment is best reflected by the spiritual equivalent of the same he wishes to push upon autists via. psychotropics?
His position about the psychotropization of autists to their certain spiritual, emotional, and intellectual deaths as to create perfect walking personified men of straw is something I’ll cover next, with respect to the actual scientific findings of an industry hell-bent on pushing monoaminergic/neurotransmission-monism with blatant disregard for long-term consequences.
PSYCHOTROPICS ARE SCIENCE NEITHER IN KOREAN NOR WESTERN DEFINITIONS
Just a reminder:
In English, science is something like this:
n. “knowledge uncovered using the scientific method* to test hypotheses in the replication of methodology.”
Chosona Korean’s equivalent for science is “gwahak”; when used with modal particle, “gwahakida” (Kim Jong-Il’s 1994 thesis title in Korean is Sahuijuuineun Gwahakida). It means something, rather, like:
“something unchanging, something perfectly developed, in reflection of the historical study in its development; something immutable as consequence of all these.”
The latter one is easy to admit defeat upon. The failure of various psychotropic regimes to effectuate change on a level on that barely countable beyond placebo in previous generations, leading to new pathways being explored, and primarily on accident rather than through a conscious awareness of it, are all, in their various degrees, implicit and explicit admissions of psychotropics to fail the test of gwahak. First, there was no perfect rectification before action, the primary Confucian-paradigm proto-scientific principle upon which most Korean science is still predicated; nobody actually knew shit before their “discovery”, so buffoonish Whites of imperfect intelligence are assigned the false recognition of “genius” for their discovery, without knowing full significance until they’re no longer alive to be accountable. Secondly, as for its implicit admission, the beneficial-adverse effects ratios were so heavily weighed upon the latter that the improvement for many was spurious in any meaningful way beyond subduing, but this is well known.
The first definition requires a little more rigour. Not because Western science is inherently more rigorous, mind you. Obfuscation is rife with the mystification and desire to make unobvious in implication the findings as to create the illusion of perfection: perfunctory attention to irrelevant fine detail at the molecular level, willing ignorance of its meaning in terms of wider, “cascade” as Ray Peat called it, ramifications for wider sects of the neuroendocrine and neurobiological systems these drugs, especially in their older generations but no less, except by degree, in their newer generations, are waging their assault upon.
Mongoloids were quite happy to keep the extent of their knowledge of euphoriant, inebriant, and other illicitly “fun” effects at the level of food. Food, in Confucianism, is considered a drug; herbs, etc. were generally subsumed under this definition rather than being considered the separate things known as ‘drugs’ in their own right today. They’d let nature take the course of the mentally ill, which would have varying positive and negative effects depending on the innate personality of that person so as such wasn’t all bad and even sparing of the more distasteful elements of society: I doubt Amber would be considered as the shaman it thinks he is, nor would I think myself to be immediately shippable off to the nearest whatever-the-precursor-to-a-work-camp was.
Amber’s reversal of that predicament of nature is predicated upon the pushing of an unnatural psychotropics agenda. It is Songbun at the level of medical (mal-)practice.
If you want to know of the ways the NHS is inherently held free of accountability by way of evasion of responsibility, read the version of Final Retrospections edited by Cuntster which remains on Scribd. Some of the original material pertaining to that is still there.
The following, in sum, is partially ethical contention, partially factual, and strongly vocal of a position in which, per the lack of intentional specificity of such treatment regimen, autism’s treatment is being sought on the basis of the presently available monaminergic intervention and the conception of which on like-paradigm (the monaminergic model); I call for its perception to be one of abhorrence. APANA or Autistic People Against Neuroleptic Abuse, admittedly slightly neurodiversitarian in leanings, before it was starved of funding by Cuntster, still had material available on scant archives about the story in which a well-functioning young woman with autism was brought to the brink of ruin just with a small dose of risperidone (a fate spared of LagoonaBlue given an agreement struck by the two of them to spare her of the spurious-in-any-case diagnosis some time in future). The main anecdote is available if one looks hard enough.
As it has so far, I do expect it to fall on deaf ears; one will remember that my first initial contention with the inherently morally abhorrent nature of drugging autistics unconsensually with antipsychotics was followed with a flower though, ultimately, empty riposte by Cuntster, advocating, in particular, SNRIs (although Venlaflaxine is more accurately an SNDRI — the D component is a little weak compared to the amphetamines or even modafinil, admittedly), adjunct to an antidopaminergic agent. Understand that I’m not using the term for the same reasons as Cuntster. I don’t agree with its reallocation from the obviously psychotic (they’re getting progestins as treatment in the next half-decade or so, cf. Galaxone) as a suppressant for all autistic intellectual traits and behavioural ones, knowingly understood of its myelin-eating, grey-matter striking properties as to literally rip through neocortcial tissue, in the same way as one would literally open up the skull and tear the tissue manually (which I’m sure is a literal fantasy of Amber’s). I just happen to disagree with the designation “antipsychotic”, as it barely possesses those properties either. As a panacea for any condition, it should be discouraged, and the autist’s self-responsibility (indeed a component of chaju) encouraged.
Whilst nothing about what Cuntster has said about the neurochemical mechanism of such a substance is inherently technically incorrect, it is understated and oversimplified as to what that is and the implications of its effects; whilst not being mechanically untrue, it is implicatively problematic. The pharmacologically heterorthodoxical perspective demonstrates illiteracy of the very newest findings, and even some of the repressed, obscure older ones.
One final caveat: yes, I overuse caffeine, knowing its adrenal effects. However, I use plenty of other things, countering them, and many of my techniques are implied in this piece. Caffeine isn’t a “dumbed down” version of the SNRI-risperidone (or whatever of the latter class of drug Amber is suggesting) protocol; it’s a knowing choice in that I’m aware of its lack of interaction with the main neurotransmitters, upregulating of choline for the enhancement of memory (antipsychotics block choline synthesis and reception), and its main stimulatory pathway of action not being dopaminergic, but rather, through the release of vasopressin. Rather, it’s by the means Amber is advocating that he wants to dumb me down, which I’ll proceed to explain in due course.
Seretonin reuptake and true affective effects
Seretonin reuptake is an awkward and only vaguely understood process. The reuptake pump of the synaptic site is blocked but only indirectly through receptor inhibition; the other steps in-between are quite vague as to their action. During the intervening period of serotonin’s accumulation, it actually has the effect of an antagonist and a depressant, and the desensitization of the neuron to serotonin’s occurrence simultaneously with the blockage of the neuron only worsens matters. There is an inherently high risk for suicide during the period and thereafter if it fails, which it does at rates near-enough placebo.
Conscious return of the emotions isn’t really anything of a consideration, unlike with the few experiments of MDMA, or what transsexuals describe on estrogen, or even what I experienced on the latter. A cheap argument in counter to this is that it is only with estrogen’s upregulation of serotonin that such effects are achieved, but we know the serotonin reuptake process isn’t anything like applicable or analogous to how either estrogen or MDMA do it. With this numbing of affect, they literally only want to recreate negroidal, inconsiderate, r-select, lumpenproletarian psychopaths of the lowest degree, extrapolating this attitude over society at large. (This is also why, despite all the estrogen in the world, Amber is still an angry little boy at heart; he lacks the neurobiological make-up for it to take the appropriate effect.)
The opposition that Cuntster thus demonstrates to the conscious return of my own emotional state at the fullest experientially available level is thus reflective of a desire to have the reputation of his non-existent empathy salvaged, whilst seeing the non-existent “robot from within” come hither forth through unnatural (de-)melioration. If he had any interest in my reformation, it wouldn’t be, if we’re going to throw categorical misnomers around, in throwing at me a proautisticant. Even in NHS reviews of drugs, this context of usage (application in autism) is particulary problematic and suggested to worsen it beyond the apparent docility imparted. Drugs used in this way, despite seemingly rectifying affective states arguably misattributed to the autist in an absence of empathy which could only be described institutionally wide in that the preponderance of neurotypicals populating their ranks are beings for whom the TOM, discussed widely already, is in a state of incompatibility rather than superiority, are deployed only for one purpose: as a confirmation of the unchangability of autism, as a confirmation of its inhumanity, and to numb the autist to their socially deprived conditions so that he is oblivious to the blatant insult being encapsulated both in-metaphor and in-actuality in such a pathetic pellet of soma. The problems with such a conception being rendered invalid under a spurious claim to synergistic action shall be discussed later, but it is a serious weighing to be considered even on its own, and for the contention to disappear with such a flippant remark of dismissal is to callously expect its ignorance to be a substituion for critique.
Cuntster even admits it himself, in an indirect way. Nothing about any statement of his regarding my mood can be considered reliable, these are second hand readings of what was an about-face of the position Haselgrove herself had harboured, that any depression was reactionary. For such cases, called adjustment disorders, the drugs are inappropriate. He mischaracterises a major depression for the sake of pathogenesis without respect to the unchangeable personality (i.e. independent of autism; “happy, oblivious retard” is an archetype only applicable to so many) dispositions of the patient, also to be discussed further in the article.
Coincidentally, serotonin as a mood-regulator in an upward director and the reliability of its effects’ direct dependency upon it are something in question. Serotonin is known to cause cortisol release by overstimulating the adrenal cortex, raising ACTH. Granting a cortisol suppressant at the enzymatic level like ketoconazone had demonstrated an irresponsiveness to SSRIs, SNRIs, etc. Euphoria from SSRIs is thus a property of the cortisol-release in part, but independent of the serotonin, which is demonstrably ineffective in the latter’s absence; cortisol, by the way, is neurodegenerative, immunosuppressive, and an aging chemical — its strongest metabolite, decadron, is used as an artificial aging agent in Pakistani girls to increase estimates of their age by investigators in illegal underage brothels. So not only does Cuntster want me to acquiesce to oblivious retardation, he wants me to look even more pig disgusting! I remember how brown I turned under the influence of Venlafaxine, for instance.
5a-reductase activation is intellectual negrification disguised as an inadvertent euphoriant
There are two pathways by which antidepressants increase testosterone, a known mood-depressant, antiempathogen, and, as I term it, intellectual negrifier, in that its strongest metabolite, dihydrotestosterone, blocks estrogen at the receptor level to induce long-term degeneration (the opposite of long-term potentiation) at the hippocampus, and general, all-round neocortical damage ala antipsychotics. One works at the level of Cytochrome P450 sites. The other, works by increasing the activity of an enzyme called 5a-reductase. The former increases free testosterone; the latter increases the rate at which that is converted from the pure compound to its more potent dihydrotestosterone metabolite form. If there is one way that antidepressants eventually lose their effectiveness, it’s by this.
In increasing the activity of 5a-reductase, another endocrine hormone is used up which arguably has antidepressant effects working indirectly at the level of its testosterone and cortisol antagonism by unbinding its association from their respective receptor sites: progesterone. Progesterone also has neurolytic qualities in its own right, extending the glial sling to create new white matter myelin networks. Progesterone converts into allopregnanolone, nothing inherently bad in this process alone (and when uninterrupted, allopregnanolone converts back into progesterone). I will discuss progesterone’s role as a serotonin neutral antagonist which disproves the seretonin hypothesis and, in my experience having used it, is the only real euthymic agent.
Too active a level of 5a-reductase activity, and progesterone starts depleting. Allopregnanolone levels struggle to keep in parallel yield, also, declining with it. Although it’s not certain yet, the jury is still very much out on whether antidepressants interrupt conversion back the way. If they do, it’ll mean a gross-deficit of progesterone and an excess of unnecessary allopregnanolone which lacks some of its wider endocrinological qualities.
The end-result is excess cortisol levels. Progesterone is needed as raw material for cortisol. However, low progesterone will also mean what little cortisol there is (though comparatively more than there would be without antidepressants as they cause the latter’s upregulation via. adrenal stimulation, remember) will bind more tightly to glucocorticoid receptors. Neurodegeneration ensues.
I’ve felt the SSRI-ineffectiveness burn-out. You’re brown, you’re mentally negroidal as fuck (brain fog, although I’ve honestly just called it “oogabooga” when otherwise-inebriated), you feel more worthless than when you started because you know the system has cheated you out of your Aryan qualities. It’s almost as if Amber knew all of this and disingenuously tried to disguise his fetish for seeing monkeys like him walk everywhere.
Norepinephrine is a stimulant of the overstressed geriatric; its presence isn’t causative of eudemonia, but coincidental to years of damage
When all is said and done to years of the natural aging cycle, simulated at an accelerated rate due to the ACTH-upregulation of SSRIs and SNRIs but which is nevertheless something that occurs in all of us unless you’re an estrofem-microgest abusing whore like Amber, norepinephrinergic neurons are the only ones left after the dopaminergic and serotonergic ones are burnt out. Your brain is basically just a soup of norepinephrine on the function level thereafter, and there’s poor interactivity of the other neurotransmitters in terms of affinity with norepinephrine receptor neurons. This form of docility is understood to be a disease (albeit itself of a natural phenomenon) conventionally, yet its iatrogenically induced simulation by neurochemical means to supress the behaviourally inconvenient isn’t seen as problematic; geriatricizing the autists and, to a certain extent still, the schizos, confining their neurocognitive function to a premature death-panel, isn’t seen as inherently wrong, because of whatever hyperbolic alternative the dumbshit pseudonegress Amber wishes to dream up at that particular time of day.
Norepineprhine reuptake inhibitors are used as ADD-medication in children, sometimes; dopaminergic agents are the preferred norm nowadays. Atomoxetine has long since given way to dextroamphetamine, but on the occasions the former is used, I can chime in as to their personal effects: it was my “babby’s first nootropic” when starting out on noots. I did feel genuinely relaxed, without the serotonin-induced cortisol-release, or the dopamine-blockade induced retardation. I felt a genuine sense of focus. It also had some pleasant sexual side-effects which may be attributable to its upregulation rather than estrogen itself in the instances an estrogenic agent is used in transsexuals; the refractory period is increased, as is the length of orgasm.
Now, this is different from an aged state in which dopamine and serotonin neurotransmission is markedly reduced, but even when this is taken into account, I doubt the sexual effects would change even if the intellectual faculties are compromised by way of this neurotransmissive deficit.
I’ve a theory, therefore, as to why hons even exist. It’s spelled in the aforementioned, in black and white!
Crosstalk synergistics between norepinephrine and serotonin don’t exist
Admittedly, Cuntster added “synergistic” as a bullshit non-word with no pragmatic application to fool everyone into thinking there was a substance behind the individual effects, mostly not cross-transmissible, of adding an anti-dopaminergic to a pseudo-proserotonergic and norepinephrinergic. The clue is that the former is in blockade of something, whilst the latter is in upregulation and part of the latter doesn’t even involve genuine upregulation (cf. why serotonin reuptake isn’t genuine antagonism). We’re not speaking of additive effects, so down the inferential chain of the definition provided by him, we can’t possibly speak of compound effects above and beyond the sum of individual effects. Drug interactions of a more superficial sort — antidopaminergics work on 5HT/serotonin receptors as well — are arguably more of a shot in the dark at the perpetuation of an unworkable serotonin-reuptake hypothesis, perpetuating the symptoms of depression down the longitudinal line of pan-endocrinological burn-out.
In the one respect we can speak of additive effects, it is in side-effects, the only thing known to be dose-dependent over many drugs, the only thing known to have a direct correlation in all of the aforementioned drug classes, and thus, the only thing to which this logic could possibly apply. It is, in any case, the thing that Amber wishes me to be disabled by, per the Final Retrospections thesis, that autism is a designation to be perpetuated of its disability rather than meliorated of its eudemonia per other conditions’ treatment.
The negroid should have his skull physically incised to access those preciously misused neurons of his, ripped apart physically as a punishment for his impudence.
Serotonin antagonists of the neutral sort are the real antidepressants
Serotonin, as has been expounded to death at this point, is not a meliorator of consciousness, emotional awareness, lexithymia, or conscious cognizance of one’s thoughts. Dependent upon itself, those processes are only dampened. It down-regulates dopamine which worsens the intellectual manifestation of the antidopaminergic anti-nootropics, as if their blockade of cholinergic action wasn’t enough. Wouldn’t it be all better if we could just… I dunno, flush it all out?
This is where progesterone comes in. It’s a serotonin neutral antagonist. Unlike inverse agonists, which compete with agonistic subtrates to prevent agonism of it to a receptor, or antagonists, which are tantamount to a receptor blockade whilst causing its post-synaptic and extracellular accumulation, neutral antagonists sit on the receptor, do nothing, and neutralise the build-up of the displaced substrate around it. Progesterone essentially causes a whole-scale seretonin antagonism both extracellularly and intracellularly, thus, “flushing” it all out, in a way similar to substances like tianeptine, an SSRE which I’ve also tried to brilliant effect. My brain was denegrified in an unprecedented way.
If we want to see self-esteem emerge from the recipient of any drug thereafter, it’s not enough that they just “feel better”, but have long-lasting intellectual improvements concomitant to that, otherwise it is meaningless, especially for those personalities for whom social endeavours are just a source of disgust. Progesterone is a neuralator, extending the glial sling to replenish the myelin sheath and expand white-matter networks. The exact opposite of what any antidopaminergic does in the long term, or any serotonin-reuptake inhibitor by way of its glucocorticoid upregulating effects.
Secondarily to this, it antagonises testosterone, whose demonic qualities in terms of neuro-generation and mood I’d discussed earlier. What a better argument for more denegrification? I can see that Amberite witch melt into the puddle of water it should’ve inevitably always disintegrated into, the wicked witch of the transsexually dominated Occident that it is.
Neurodegenerative qualities of antipsychotics and antidepressants
Moncrieff (sp?) did some work on the former, the latter is entirely my speculation which only wraps up in an encapsulatory summary the reasoning from before. The neurodegenerative effects of antidopaminergics on both the grey and white matter volume are quite well-established over a series of studies. The main riposte in critique of the proclamation that it is aetiologically accountable to the disease itself is that over the longitudinal course, there is neither improvement of this situation on antidopaminergics, nor a more rapid rate of degradation.
When we look at the reasoning from a Peatist perspective, where I’ve learned most of what I know about endocrine and neuroendocrine biology, it becomes clear to us. A dopamine blockade causes an accumulation of dopamine; newer drugs imperfectly blockade it by rapidly associating and disassociating from the receptor, causing a burnout. Dopamine’s extracellular presence is inversely correlated with intelligence — the reckoning behind this is less due to the presence of dopamine itself, and more due to its interaction with the wrong kind of receptors, resulting in the wrong connections (in the inferential chain of neurotransmitter interaction resulting in influences upon the directionality of glial sling; hormones, again, do this more powerfully still), nothing a whole-scale blockade really does anything other than worsen in the long-term; keep in mind, not everyone who has high levels of extracellular dopamine is a retard, for when one has endogenously high progesterone, it’s utilised better.
In a low-progestin environment with high glucocorticoids, high seretonin crosstalk interactivity, and high androgen levels, that dopamine is basically useless, blockaded from pet-receptor sites of the adherents of stupid, antiquainted schizophrenogenic hypotheses, or not. Studies into schizophrenics show that the endocrine burn-out had long preceded the neuroendocrine one, manifestations on a neurochemical or neurobiological level, and the last stage of this chain (in the powerful exertion of behavioural control by the willingness of some), the manifestation of symptoms themselves.
Social approachability from making self-unaware retards? I think not.
I’ve seen the retards sludging their way through the corridors of autism support organizations when I used to attend them long ago. Many of them were on some kind of psychotropic regime of the variant to Amber’s suggestion (I did have the good sense to ask those who were even able to speak to any sort of level resembling normal-range human speech). Their consciousness was visibly compromised in a way as to make the beneficial-adverse effect ratio’s weighing to the latter obvious. I would, honestly, rather have been situated in the “fat, drugged up psychopaths eating chocolates” scenario of Ronson’s work, because at least there, they had the neurobiological make-up to feign at an advanced level the trappings of human behaviour to a high level, which these autists had lacked, and were being compromised further of their ability to do.
Amber isn’t oblivious to all of this. He intentionally wishes to worsen the social meliorability of autists to get across an agenda; the deservingness of their neglect as they walk their proverbial death-rows to the death panel of a psychotropically induced, premature mental death via. dementia. To somebody undrugged and free-range like myself who had a very socially conscious awareness of how one had represented themselves in the presence of these iatrogenically-autistified ‘tards (wasted on them, in retrospect of the realisation they were devoid of interpreting cue, of course), this didn’t make them any more endearing to me. I wanted to avoid especially the socially hyperactive ones driven on the kind of regimes probably prescribed per Amberite fashion.
They were made insensitive, obnoxious, stupid hypermales, from an infrastructure in which those traits’ previous presence were more than doubtful, per the anecdote of the founder of APANA whom had regaled risperidone’s effects on her daughter.
Paranoia is cultural when its source originates from perspectives of experience specific to the organic situational “badness” of the disenfranchised
The last thing psychotherapy is going to do is improve the situation when the drugged autist interacts (I use this in a sociological context, the one Amber likes to ignore) with the prejudices of the practitioner to result in the presumption on the latter’s end that they were always this obnoxious, insensitive, boorish and beastial. If there is any sort of cognizance left on the autist’s part, they will pick up on this and disengage. If there isn’t, one can’t expect the autist to be anything near articulate enough to contribute in any wise or meaningful way to the psychotherapeutic process of mutual dialogue, and s/he is left short changed.
I can speak with some experience to the former situation, and it was without drugs having caused the production of inappropriate social cues resulting in their misinterpretation. My anxiety (if it were food, I’d have enough to feed all of Amber’s starving ancestors in the Kenyan outback) was misinterpreted as anger; a theme Haselgrove has since purposefully run with, though this was with a bit of a duller psychotherapeutic-model type for whom this misapprehension can be assumed to be sincere. That a study would come a year later (my psychotherapy sessions took place in 2015; the study was published 2016 — my own suspicions as to the hypothesis however have been something going back to my extremely introspective adolescence) to confirm a fundamental incompatibility at the level of such an interactive process with two different forms of mind-theoretical make-up would be sheer luck, though Amber has only since run with it again, to be abused to his benefit.
Autistics don’t have a nuanced differentiation between fight and flight, I reckon. In credit to Amber’s demeaning trivialization of all features of autistics to be likable to children, in this one respect, it would be in accordance with such a model. An autist is afraid? They’ll fight it. An autist is angry? They’ll fight it. I’ve noted it with myself.
Because, in a world where I’ve experienced nothing but the antipathy of neurotypicals even preceding my encounters with psychiatry, I have only ever known the neurotypical to be an adversary, a hostile party, a traitor, a psychopath, a manipulator, a demon, the beast I should conquest by taming it, through force if necessary. Would you want to show fear in front of such bastards? Instinctively no! Beat that Ambeirte demon up no matter what it is you actually feel, and you show a sense of formidability that would otherwise not invite sympathy had you done the opposite, but snivelling disdain, exactly as the psychopath does, to any display of weakness.
Does Cuntster really want an individual’s autism to improve?
If that were the case, not only would he haven’t have misdirected the conversation to some sort of categorical imperative for their eradication justifying a withholding of basic resources from them, he wouldn’t so callously advocate a drug regime which experientially not only has ended in failure or use specious noise-constructs (“muh synergism”) to distract the attention away from his misrepresentation and scapegoating some unspoken, non-existent misunderstanding on my end, but whose stated (marketed) effects are better supplanted by alternative substances procured by the grey-market.#
Categorically, I’m thoroughly unconvinced that this is the case.
This is more disgusting than any of the supposed “atrocities” misattributed in abhorrently perverted historical revisionism to the regime of Chosun. Amber’s relationship with me is an actual microcosm, of the harm mischaracterised of the Kim dynasty to his people and the narcissistic thrill of power he derives from it.